Endless Forms Most Beautiful: The New Science of Evo Devo by Sean B. Carroll, Jamie W. Carroll, Josh P. Klaiss

By Sean B. Carroll, Jamie W. Carroll, Josh P. Klaiss

Author note: Jamie W. Carroll (Illustrator), Josh P. Klaiss (Illustrator)
Publish 12 months note: First released April eleventh 2005
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“A attractive and extremely vital book.”— Lewis Wolpert, American Scientist

Throughout a lot of the animal or even plant kingdoms, virtually an identical old genes code for nearly an identical proteins. and extra, to everyone's astonishment, the genes curious about making the advanced eyes of fruit flies are shut fits to these curious about making the very various eyes of octopuses and folks. So what ends up in the nature's "endless kinds so much beautiful"?

The key to this secret is being unravelled through "Evo Devo" or the hot technological know-how of evolutionary improvement biology. by means of how a single-celled egg supplies upward push to a fancy, multi-billion celled animal, Evo Devo is illuminating precisely how new species – butterflies and zebras, trilobites and dinosaurs, apes and people – are made and developed. the main, it seems, is all approximately position and timing... For somebody who has ever reflected the place did I come from?, never-ending varieties most pretty explores our heritage, either the adventure we've all made of egg to grownup, and the lengthy trek from the beginning of existence to the very fresh foundation of our species.

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Extra info for Endless Forms Most Beautiful: The New Science of Evo Devo and the Making of the Animal Kingdom

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The provirus itself possesses a large block ­of duplicated sequence at each end of the virus sequence. These direct repeats may be several hundred to a thousand bases long. 1. Structural genes common to ­retroviruses Gene Full name Function of protein products gag Packages viral RNA prt pol group-­specific antigen protease polymerase env envelope Processes viral proteins A multi-­f unctional protein with endonuclease, RNA-­dependent DNA polymerase (that is, reverse transcriptase) and RNA-­degrading activities A viral membrane protein that mediates viral adhesion to cells; suppresses immunity are called long terminal repeats (LTRs), and they contain the DNA sequence motifs needed to regulate viral gene expression.

In this case, two outcomes may follow the exchange of genetic material. 12, upper diagram). 12. Homologous recombination between different ERVs of the same t y pe Equal recombination is shown in upper diagram; unequal recombination in lower diagram. chromosomal DNA that extends for an indeterminate distance beyond the ERV. In the absence of a compensating recombination event, the result would be a chromosome translocation. Unequal homologous recombination, say between the downstream (right­hand or 3′) ­LTR of one ERV and the upstream (left-­hand or 5′) LTR of another, leads to very distinctive products.

Evidence was provided that humans share some inserts also with NWMs [25]. The shape of the family tree ­revealed by these analyses is congruent with that developed over the years on the basis of a whole range of other criteria. But even if we had never heard of evolution and knew nothing of taxonomy, discovery of the relationships established by patterns of ERV insertions would have compelled us to propose an evolutionary theory of common descent, along the lines that taxonomists have laboured to develop over many years.

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